Meet Dr. Siragy
Dr. Helmy M. Siragy, M.D.,
Professor of Medicine and Endocrinology
University of Virginia
Dr. Helmy Siragy is a Professor of Medicine in the Division of Endocrinology and Metabolism and member of the Cardiovascular Research Center at the University of Virginia Health System in Charlottesville, Virginia, where he is also an attending physician at the University of Virginia Hospital.
Dr. Siragy obtained his residency training at the University of Texas Medical Branch in Galveston, TX, followed by fellowships in Endocrinology and Hypertension under the supervision of Professor Frederic C. Bartter at the University of Texas Health Sciences Center in San Antonio. Dr. Siragy is board certified in internal medicine, with subspecialty in endocrinology, diabetes and metabolism. He is also certified as Hypertension Specialist by the the American Society of Hypertension.
Dr. Siragy is a fellow of the American College of Physician, the American Heart Association and is a member of numerous national and international Endocrine and Hypertension Societies. He is a reviewer for the American Heart and the National Institute of Health Study Sections and several national and international medical journals. He sets on the editorial board of The Journal of Hypertension, the official journal of the International Society of Hypertension and is the Section Editor for the Antihypertensive Agents: Mechanisms of Drug Action section, Current Hypertension Reports.
An international lecturer on subjects related to hypertension and endocrinology, Dr. Siragy most recently was first to discover and publish that stimulation of angiotensin AT2 receptor leads to generation of nitric oxide through stimulation of Bradykinin-Nitric Oxide-cGMP cascade.
Dr. Siragy has authored more than 150 medical journal articles and twenty one medical textbook chapters, including the chapter on the kidney and the renin angiotensin system in Braunwald Atlas of Heart Diseases, Angiotensin and Diabetic Nephropathy in Harrison's online textbook of Medicine and The Angiotensin Receptor: AT1